8-2-4　ⓔ文献

  1. Bergmann O, Bhardwaj RD, et al: Evidence for cardiomyocyte renewal in humans. Science, 2009; 324: 98–102.

  2. Maillet M, van Berlo JH : Molecular basis of physiological heart growth: fundamental concepts and new players. Nat Rev Mol Cell Biol, 2013; 14: 38–48.

  3. Yazaki Y, Raben MS: Cardiac myosin adenosinetriphosphatase of rat and mouse. Distinctive enzymatic properties compared with rabbit and dog cardiac myosin. Circ Res, 1974; 35: 15–23.

  4. Komuro I, Yazaki Y: Control of cardiac gene expression by mechanical stress. Annu Rev Physiol, 1993; 55: 55–75.

  5. Schiattarella GG, Hill JA: Inhibition of hypertrophy is a good therapeutic strategy in ventricular pressure overload. Circulation, 2015; 131: 1435–1447.

  6. Levy D, Garrison RJ, et al: Prognostic implications of echocardiographically determined left ventricular mass in the Framingham Heart Study. N Engl J Med, 1990; 322: 1561–1566.

  7. Simpson P, McGrath A, et al: Myocyte hypertrophy in neonatal rat heart cultures and its regulation by serum and by catecholamines. Circ Res, 1982; 51: 787–801.

  8. Komuro I, Kaida T, et al: Stretching cardiac myocytes stimulates protooncogene expression. J Biol Chem, 1990; 265: 3595–3598.

  9. Nakamura M, Sadoshima J: Mechanisms of physiological and pathological cardiac hypertrophy. Nat Rev Cardiol, 2018; 15: 387–407.

  10. Knoll R, Hoshijima M, et al: The cardiac mechanical stretch sensor machinery involves a Z disc complex that is defective in a subset of human dilated cardiomyopathy. Cell, 2002; 111: 943–955.

  11. Eder P, Molkentin JD: TRPC channels as effectors of cardiac hypertrophy. Circ Res, 2011; 108: 265–272.

  12. Zou Y, Akazawa H, et al: Mechanical stress activates angiotensin II type 1 receptor without the involvement of angiotensin II. Nat Cell Biol, 2004; 6: 499–506.

  13. Krumholz HM, Larson M, et al: Prognosis of left ventricular geometric patterns in the Framingham Heart Study. J Am Coll Cardiol, 1995; 25: 879–884.

  14. Opie LH, Commerford PJ, et al: Controversies in ventricular remodelling. Lancet, 2006; 367: 356–367.

  15. Sano M, Minamino T, et al: p53–induced inhibition of Hif–1 causes cardiac dysfunction during pressure overload. Nature, 2007; 446: 444–448.

  16. Oka T, Akazawa H, et al: Angiogenesis and cardiac hypertrophy: maintenance of cardiac function and causative roles in heart failure. Circ Res, 2014; 114: 565–571.

  17. Gibb AA, Hill BG: Metabolic Coordination of Physiological and Pathological Cardiac Remodeling. Circ Res, 2018; 123: 107–128.

  18. Delbridge LMD, Mellor KM, et al: Myocardial stress and autophagy: mechanisms and potential therapies. Nat Rev Cardiol, 2017; 14: 412–425.

  19. Henning RH, Brundel B: Proteostasis in cardiac health and disease. Nat Rev Cardiol, 2017; 14: 637–653.

  20. Mann DL: Inflammatory mediators and the failing heart: past, present, and the foreseeable future. Circ Res, 2002; 91: 988–998.